The air is therefore trapped in the lungs, making it hard for the lungs to send the right amount of oxygen to the rest of the body (McCance & Huether, 2019). rada jones md 09/12/06. Free access to premium services like Tuneln, Mubi and more. COPD - . Tap here to review the details. Looks like youve clipped this slide to already. what is, COPD - . -System-wide nurse referral bonus program - earn up to $6000 per referral. Want to work with us? cydnee patterson, ali lubbers, nicole newinger , heidi proudfoot & kaitlin oudshoorn. Each of the nine Guides for Better Living focuses on one important aspect of life with COPD. Awareness and compliance with Guidelines for the management of Stable COPD in Current Outpatient practices and patients' knowledge of COPD - Active Detection of Chronic Obstructive Pulmonary Disease and Asthma in the General Population Vol 158. pp 1730 1738, 1998. The SlideShare family just got bigger. Many in this series of 12- to 20 . Number of Views: 87. 1.) Now customize the name of a clipboard to store your clips. Many in this series of 12- to 20-page easy-to-read booklets feature interactive portions for patients and caregivers to discuss together. COPD - Physical Findings Tachypnea Accessory respiratory muscle use Pursed lip exhalation Weight loss due to poor dietary intake and excessive caloric expenditure for work of breathing, Dominant Clinical Forms of COPD Pulmonary emphysema Chronic bronchitis Most patients exhibit a mixture of symptoms and signs, COPD - Advanced Dx secondary polycythemia cyanosis tremor somnolence and confusion due to hypercarbia Secondary pulmonary HTN w or w/o cor pulmonale, COPD Treatment Strategy Elimination of extrinsic irritants bronchodilator & glucocorticoid therapy Antibiotics Mobilization of secretions respiratory vaccines Oxygen therapy - if oxygen saturation <90% at rest on room air, A-a gradient A-a gradient = predicted pO2 observed PO2 PAO2 = (FIO2 X 713) (PaCO2/0.8) at sealevel PAO2 = 150-(PaCO2/0.8) at sealevel on room air Normal range 10-15mm > 30 years of age Normal range 8mm < 30 years of age Increased A-aDO2=diffusion defect Right to left shunt V/Q mismatch, Examples A doubel overdose brings two 30 yr old patients to the ED. By whitelisting SlideShare on your ad-blocker, you are supporting our community of content creators. Avg rating:3.0/5.0. review oct. 16, 2014 cathy vakil. We've updated our privacy policy. Objective data is also assessed. Chronic Obstructive *ctda0&?iOcdr0&O@(L%EUxmsy]r@y9`P Appointments & Locations. Scavenger Hunt Week 3, Choice 1 Here finally is your chance to see The many facets of creativity; Theories, presentations, lessons & such Without reality are nothing much. Goal: Develop a PowerPoint presentation on a cardiovascular/pulmonary disorder/disease discussed in the McCance text. You may decide to use one or two spots . Pneumothorax is one of the disorders of the chest and lower respiratory tract. definition. Nurse CHI Pulmonary and Sleep Medicine Jan 2022 - Present 1 year 3 months. This is an original nursing presentation for PowerPoint. View (Order 4698406) Powerpoint Presentation on Obesity (2).pptx from ENG 246 at North Carolina State University. Learn faster and smarter from top experts, Download to take your learnings offline and on the go. copd. Blood gases provide additional information about alveolar ventilation and acidbase status by measuring arterial PCO 2 and pH. courtney pearce & jenna paterno . . Contact Hours: 42.5 This nursing continuing professional development activity was approved by the American Association of Critical-Care Nurses, an accredited approver by the American Nurses Credentialing Center's Commission on Accreditation. Asthma patients can also develop chronic (not fully reversible) airflow obstruction. 7pm. . COPD- pulmonary hyperinflation- the diaphragms are at the level of the eleventh posterior ribs and appear flat. Updated November 2021. Normally, the airways and air sacs in your lungs are elastic or stretchy. It appears that you have an ad-blocker running. Do not sell or share my personal information, 1. CHANGE IN COLOUR OF SPUTUM . Our beautiful, affordable PowerPoint templates are used and trusted by both small and large companies around the world. It appears that you have an ad-blocker running. By: DrDwayne California University Balance Between Formal Teams Communities of Practice Re California State University Light a Candle or Curse the Darkness.docx, California State University Utilitarianism Kantian Ethics Videos Questions.docx. briefly review the gold classification of copd, COPD - . Continuation Obese patients also need increased physical exercises monitored by nurses. COPD further branches into three specific lung conditions: emphysema, chronic bronchitis, and refractory asthma. The app. Download your copy here or use the interactive My COPD Action Plan in the COPD Pocket Consultant Guide (PCG) app (free for download in the App Store and Google Play). Blood gases drawn on room air revealed these values: patient 1- pH =7.18, PCO2 = 70mmHg, PO2=50mmHg, HCO3=24mEq/L; patient2- pH =7.31, PCO2=50mmHg, PO2=50mmHg, HCO3=25mEq/L, Comment The A-a gradient calculation for patient 1 is as follows: A-a DO2 = PAO2 PaO2 PAO2 = 150 (1.25x PCO2) PAO2 = 150 (1.25x 70) PAO2 = 62 A-a =62 50 A-a = 12. CrystalGraphics, Inc. 1999 S. Bascom Avenue Suite 700 Campbell, CA 95008 USA. Looks like youve clipped this slide to already. 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Views: 670, By: DrDwayne 2023 Spring Consortium: On-Demand. Designed to support NPs in primary care who play a crucial role in the management of patients with PAH, a rare, complex disease this tool explains the clinical presentations of PAH, provides an overview of treatment options and identifies when patients should be referred to pulmonary hypertension (PH) centers. It appears that you have an ad-blocker running. It has a simple style, with a white background and light blue waves and lines, which convey elegance and serenity. Electrocardiography may detect signs of ventricular hypertroph, Classification GOLD stage Classification based on pathologic type, 2023 SlideServe | Powered By DigitalOfficePro, - - - - - - - - - - - - - - - - - - - - - - - - - - - E N D - - - - - - - - - - - - - - - - - - - - - - - - - - -. An X-ray can also rule out other lung problems or heart failure. Citation: Gundry S (2019) COPD 1: pathophysiology, diagnosis and prognosis. The air buildup puts pressure on the lung (s), so it cannot expand as much as it normally. Chronic Obstructive Pulmonary Disease (COPD) Chronic obstructive pulmonary disease (COPD), which includes chronic bronchitis and emphysema, is a long-term lung disease that makes it hard to breathe. Instant access to millions of ebooks, audiobooks, magazines, podcasts and more. Smoking is the most common cause. The Guides are available for purchase individually or in a set in our online catalogue. Presentation on Obesity Name Course Date Introduction Obesity is the excess of . Enjoy access to millions of ebooks, audiobooks, magazines, and more from Scribd. Energy-conserving tips are presented. Copyright 2023 Freepik Company S.L. -System-wide nurse referral bonus program - earn up to $6000 per referral. However, people whose disease progresses to more severe stages . COPD (chronic obstructive pulmonary disease) is a group of lung diseases that make it hard to breathe and get worse over time. June 2023. The NEW! Views: 1320. pulmonary diseases. Enjoy access to millions of ebooks, audiobooks, magazines, and more from Scribd. Send us a message and help improve Slidesgo. The COPD Foundation Disaster Preparedness Plan (DPP) is a comprehensive resource for individuals and families impacted by chronic lung diseases, including COPD. California State University how To Reduce Potential Vulnerabilities Discussio California State University Long Beach IRAC Business Law Discussion.docx, CALUMS Personal Statement for University Application Admission Essay.docx. This flyer will give you helpful tips and tools for staying healthy, avoiding illness, and recognizing early warning signs of an infection or flare-up. Weve updated our privacy policy so that we are compliant with changing global privacy regulations and to provide you with insight into the limited ways in which we use your data. Chronic obstructive pulmonary disease ppt, Chronic obstructive pulmonary disease by aminu arzet, CODP ( Chronic Obstructive Pulmonary Disease ), COPD - Chronic obstructive pulmonary disease - Aby, Pharmacotherapy of Chronic Obstructive Pulmonary Disease, Chronic Obstructive Pulmonary Disease (COPD). Chronic Obstructive Pulmonary Disease - COPD. For health care professionals, hard copies of the COPD Pocket Consultant Guide, including the treatment decision tree, seven COPD severity domains, the CAT, the mMRC Breathlessness Scale, and listings of brand or generic medications are available for download below. Learn about tools to help you quit smoking as well as risk factors for COPD in this double-sided flyer. Bandaged Teddy Bear - Nursing Templates Free. The disease affects millions of Americans and is a leading cause of disability and death in the U.S. The presentation must educate advanced practice nurses on assessment and care/treatment, including . Free access to premium services like Tuneln, Mubi and more. Ventolin) MDI or nebulizer preferred Anticholinergics (e.g. Click here to review the details. The SlideShare family just got bigger. Most patients have elements of each. working around certain kinds of chemicals, COPD - . Chronic Bronchitis Considerations Problems other than COLD should be suspected when hypoxemia is difficult to correct with modest levels of supplemental oxygen. 527 Views Download Presentation. Chronic Obstructive Pulmonary Disease (Copd), Copd(chronic obstructive pulmonary disease), treatment Chronic Obstructive Pulmonary Disease. Chronic Obstructive Pulmonary Disease (COPD) is a condition of chronic dyspnea with expiratory airflow limitation that does not significantly fluctuate. California State Musculoskeletal Metabolic and Multisystem Health Dysfunction Callimbah is a successful company that is involved in the.docx, California State University Economists and Finance of Tesco Presentation.docx. Aim: To describe for areas of improvement in the management of COPD and reduction in emergency department presentations in Queensland. ss visser, pulmonology internal medicine up. Clipping is a handy way to collect important slides you want to go back to later. 28-7, Emphysema Pathophysiology Hyperinflation of alveoli Destruction of alveolar walls Destruction of alveolar capillary walls Narrowed airways Loss of lung elasticity, Emphysema Pathophysiology Two types: Centrilobular (central part of lobule) Most common Panlobular (destruction of whole lobule) Usually associated with AAT deficiency, Emphysema Pathophysiology Structural changes are: Hyperinflation of alveoli Destruction of alveolar capillary walls Narrowed, tortuous small airways Loss of lung elasticity, Emphysema Pathophysiology Small bronchioles become obstructed as a result of Mucus Smooth muscle spasm Inflammatory process Collapse of bronchiolar walls Recurrent infections production/stimulation of neutrophils and macrophages release proteolytic enzymes alveolar destruction inflammation, exudate, and edema, Emphysema Pathophysiology Elastin and collagen are destroyed Air goes into the lungs but is unable to come out on its own and remains in the lung Causes bronchioles to collapse, Emphysema Pathophysiology Trapped air hyperinflation and overdistention As more alveoli coalesce, blebs and bullae may develop Destruction of alveolar walls and capillaries reduced surface area for O2 diffusion Compensation is done by increasing respiratory rate to increase alveolar ventilation Hypoxemia usually develops late in disease, Emphysema Clinical Manifestations Dyspnea Progresses in severity Patient will first complain of dyspnea on exertion and progress to interfering with ADLs and rest, Emphysema Clinical Manifestations Minimal coughing with no to small amounts of sputum Overdistention of alveoli causes diaphragm to flatten and AP diameter to increase, Emphysema Clinical Manifestations Patient becomes chest breather, relying on accessory muscles Ribs become fixed in inspiratory position, Emphysema Clinical Manifestations Patient is underweight (despite adequate calorie intake), Chronic Bronchitis Pathophysiology Pathologic lung changes are: Hyperplasia of mucus-secreting glands in trachea and bronchi Increase in goblet cells Disappearance of cilia Chronic inflammatory changes and narrrowing of small airways Altered fxn of alveolar macrophages infections, Chronic Bronchitis Pathophysiology Chronic inflammation Primary pathologic mechanism causing changes Narrow airway lumen and reduced airflow d/t hyperplasia of mucus glands Inflammatory swelling Excess, thick mucus, Chronic Bronchitis Pathophysiology Greater resistance to airflow increases work of breathing Hypoxemia and hypercapnia develop more frequently in chronic bronchitis than emphysema, Chronic Bronchitis Pathophysiology Bronchioles are clogged with mucus and pose a physical barrier to ventilation Hypoxemia and hypercapnia d/t lack of ventilation and O2 diffusion Tendency to hypoventilate and retain CO2 Frequently patients require O2 both at rest and during exercise, Chronic Bronchitis Pathophysiology Cough is often ineffective to remove secretions because the person cannot breathe deeply enough to cause air flow distal to the secretions Bronchospasm frequently develops More common with history of smoking or asthma, Chronic Bronchitis Clinical Manifestations Earliest symptoms: Frequent, productive cough during winter Frequent respiratory infections, Chronic Bronchitis Clinical Manifestations Bronchospasm at end of paroxysms of coughing Cough Dyspnea on exertion History of smoking Normal weight or heavyset Ruddy (bluish-red) appearance d/t polycythemia (increased Hgb d/t chronic hypoxemia)) cyanosis, Chronic Bronchitis Clinical Manifestations Hypoxemia and hypercapnia Results from hypoventilation and airway resistance + problems with alveolar gas exchange, COPD Complications Pulmonary hypertension (pulmonary vessel constriction d/t alveolar hypoxia & acidosis) Cor pulmonale (Rt heart hypertrophy + RV failure) Pneumonia Acute Respiratory Failure, COPD Diagnostic Studies Chest x-rays early in the disease may not show abnormalities History and physical exam Pulmonary function studies reduced FEV1/FVC and residual volume and total lung capacity, COPD Diagnostic Studies ABGs PaO2 PaCO2 (especially in chronic bronchitis) pH (especially in chronic bronchitis) Bicarbonate level found in late stages COPD, COPD Collaborative Care Smoking cessation Most significant factor in slowing the progression of the disease, COPD Collaborative Care: Drug Therapy Bronchodilators as maintenance therapy -adrenergic agonists (e.g. Has a simple style, with a white background and light blue waves lines... 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